Empirical claims

• Lesions to link structures (e.g., medial temporal lobe) produce

  • correlated AA and RA, with
  • the RA showing a Ribot gradient

• Discrete lesions to modulatory structures (e.g., basal forebrain) produce AA without RA

• Lesions involving both the link and modulatory system produce lowly correlated AA and RA

• Only strong patterns are consolidated